Researchers led by Dr Richard Rainbow from the University's Department of Cardiovascular Sciences have shown that blood vessels contract more strongly at raised glucose levels than at 'normal physiological' levels. Blood vessels contract and relax to control blood pressure. In general, the more contracted the GlycoCares Wellness Guidedaily blood sugar support vessels are, the higher the blood pressure. Using electrophysiology and myography techniques to examine the impact of glucose on arterial myocytes, cells that make up the tissue of our Glyco Care blood sugar support vessels, the team has identified a mechanism that controls the narrowing of blood vessels. The research comes as MPs and health experts debate proposals for a 'sugar tax' and highlights the potential health risks of consuming large amounts of rich, sugary foods regularly in your diet. With healthy eating among the most common New Year's resolutions, it adds another incentive to reduce our intake of these foods all year round. Heart attacks occur when a coronary artery, which provides the blood to the heart muscle to give the required nutrients and oxygen, are blocked.

High glucose at the time of heart attack could make this block more severe by causing the blood vessel to contract, leading to a higher risk of complications. This was an experimental lab study which means that we can draw conclusions about cause and effect in a controlled environment. Here, we have identified a known signalling protein family, protein kinase C, is a key part of this enhanced contractile response, and have also shown in our experiments that we can restore the normal level of contractile response, and reverse the effects on the heart, with inhibitors of these proteins. A large number of people who suffer a heart attack will have high glucose due to the 'stress response'. Our studies show that glucose has an important physiological effect on the normal functioning of the cardiovascular system. Increases in blood sugar to pathophysiological levels cause marked changes in normal blood vessel and cardiac muscle behaviour that could be life-threatening if left untreated.

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